Although vitamin D is synthesised in the skin through sun exposure, and is also acquired via a few dietary sources, insufficient sun exposure and low intake of dietary sources of vitamin D have led to global widespread vitamin D deficiency, linked to an elevated risk of various cancers, mainly those of the digestive system and some types of leukemia.
“For years, my lab has been dedicated to studying the molecular mechanisms of vitamin D in human cancer cells, particularly its role in stopping their proliferation,” said Prof. White. “We discovered that vitamin D controls both the rate of production and the degradation of cMYC. More importantly, we found that vitamin D strongly stimulates the production of a natural antagonist of cMYC called MXD1, essentially shutting down cMYC function.”
In an animal study, scientists found that vitamin D applied to mouse skin decreased cMYC levels and activity, and that mice lacking the vitamin D receptor had significantly elevated cMYC, suggesting that topical vitamin D may be as effective as oral forms in preventing cancer-related processes.
These results emphasize the considerable promise of vitamin D as a cancer prevention agent, and highlight its general importance in health and disease prevention.
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